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The most common cause of BPPV is head trauma. Other factors in the development of the disease include physical inactivity (sedentary lifestyle), alcoholism, serious operations in the head area, and diseases of the central nervous system. In general, there are many reasons for the occurrence of BPPV. The following are distinguished by frequency. Symptoms of benign paroxysmal positional vertigo.

A classic episode of BPPV begins suddenly with a change in body position and tilt of nolvadex pills towards the affected ear. After provoking movements, the patient begins a pre-attack period, which lasts less than five seconds. Then the attack itself begins - severe rotational dizziness. At this moment, the patient feels as if he is being thrown towards the affected ear. A person remains in this state for 30 seconds to a minute, after which the state of health returns to normal until the next provoking head movement.

The first attack of rotational vertigo for many occurs in the morning after waking up, when they try to get tamoxifen of bed and sit down. This is due to changes in the position of the head and its rotation. However, if the patient, for example, gets up from the chair without moving his head, then the attack will not start.

BPPV manifests differently in everyone. minimal movement may cause nausea and vomiting in some patients, while in others the symptoms will be milder. In both cases, hearing loss, tinnitus, headache or other symptoms are not observed. And if the patient avoids provoking movements, then there are no symptoms at all.

During attacks, specific oscillatory eye movements occur - nystagmus. By assessing these movements, the side of tamoxifen pills ear lesion is determined. During the period between attacks, when the position of the head is fixed, there is a feeling of cloudiness and fog in the head. Pathogenesis of benign paroxysmal positional vertigo.

On both sides at the end of these semicircles there areThere is a wide ampoule in which the sensing apparatus is located. There is liquid inside it. Each ampoule at the end has a cupule-like substance (gelatin-like substance) that covers the hairs of the receptors. When a person turns his head, a three-stage reaction is triggered. the movement of the fluid leads to the movement of the cupula, and the movement of the cupula leads to the movement of the receptor hairs, which transmit a nerve signal to the brain about turning the head.

To buy nolvadex online understand the pathogenesis of BPPV, it is necessary to understand the structural features of the vestibular apparatus. Normally, the vestibular apparatus consists of three semicircles, which are aligned in three different planes at right angles to each other.

    Inside the vestibular apparatus there are additional crystals - otoliths. Their particles - otoconia - are the main culprits in the development of BPPV. When the position of the body changes and under the force of gravity, they also begin to move and create pathological signals, irritating the receptors. This causes the person to feel as if they are still moving - an episode of acute rotational vertigo. This feeling does not go away until the particles precipitate. In order for them to leave the semicircular canals, special techniques are required.

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    Another, slightly less common mechanism for the occurrence of BPPV is the attachment of otoliths or parts thereof to the cupula of the semicircular canal. Because of this, constant irritation of the receptors occurs when the body position changes - cupulolithiasis.

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